Selective loss of type I interferon-induced STAT4 activation caused by a minisatellite insertion in mouse Stat2-Reference-Cited by-同舟云学术

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Selective loss of type I interferon-induced STAT4 activation caused by a minisatellite insertion in mouse Stat2

Published:2000-07 Issue:1 Volume:1 Page:65-69
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Farrar J. David,Smith Janice D.,Murphy Theresa L.,Leung Stewart,Stark George R.,Murphy Kenneth M.

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/ni0700_65.pdf

Reference44 articles.

1. Abbas, A. K., Murphy, K. M. & Sher, A. Functional diversity of helper T lymphocytes. Nature 383, 787–793 (1996).

2. Trinchieri, G. Interleukin-12: a proinflammatory cytokine with immunoregulatory functions that bridge innate resistance and antigen-specific adaptive immunity. Annu. Rev. Immunol. 13, 251–276 (1995).

3. Hsieh, C.-S et al. Development of Th1 CD4+ T cells through IL-12 produced by Listeria-induced macrophages. Science 260, 547–549 (1993).

4. Jacobson, N. G. et al. Interleukin 12 signaling in T helper type 1 (Th1) cells involves tyrosine phosphorylation of signal transducer and activator of transcription (Stat)3 and Stat4. J. Exp. Med. 181, 1755–1762 (1995).

5. Unanue, E. R. Studies in listeriosis show the strong symbiosis between the innate cellular system and the T-cell response. Immunol. Rev. 158, 11–25 (1997).

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