Helicobacter pylori CagA targets PAR1/MARK kinase to disrupt epithelial cell polarity-Reference-Cited by-同舟云学术

Helicobacter pylori CagA targets PAR1/MARK kinase to disrupt epithelial cell polarity

Published:2007-05 Issue:7142 Volume:447 Page:330-333
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Saadat Iraj,Higashi Hideaki,Obuse Chikashi,Umeda Mayumi,Murata-Kamiya Naoko,Saito Yasuhiro,Lu Huaisheng,Ohnishi Naomi,Azuma Takeshi,Suzuki Atsushi,Ohno Shigeo,Hatakeyama Masanori

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature05765.pdf

Reference30 articles.

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2. Segal, E. D., Cha, J., Lo, J., Falkow, S. & Tompkins, L. S. Altered states: involvement of phosphorylated CagA in the induction of host cellular growth changes by Helicobacter pylori. Proc. Natl Acad. Sci. USA 96, 14559– 14564 (1999)

3. Higashi, H. et al. SHP-2 tyrosine phosphatase as an intracellular target of Helicobacter pylori CagA protein. Science 295, 683– 686 (2002)

4. Higashi, H. et al. Biological activity of the Helicobacter pylori virulence factor CagA is determined by variation in the tyrosine phosphorylation sites. Proc. Natl Acad. Sci. USA 99, 14428– 14433 (2002)

5. Hatakeyama, M. Oncogenic mechanisms of the Helicobacter pylori CagA protein. Nature Rev. Cancer 4, 688– 694 (2004)

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