Inflammatory Skin Disease in K5.hTGF-β1 Transgenic Mice Is Not Dependent on the IL-23/Th17 Inflammatory Pathway-Reference-Cited by-同舟云学术

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Inflammatory Skin Disease in K5.hTGF-β1 Transgenic Mice Is Not Dependent on the IL-23/Th17 Inflammatory Pathway

Published:2009-10 Issue:10 Volume:129 Page:2443-2450
ISSN:0022-202X
Container-title:Journal of Investigative Dermatology
language:en
Short-container-title:Journal of Investigative Dermatology

Author:

Fitch Erin L.,Rizzo Heather L.,Kurtz Stephen E.,Wegmann Keith W.,Gao Wei,Benson Jacqueline M.,Hinrichs David J.,Blauvelt Andrew

Publisher

Elsevier BV

Subject

Cell Biology,Dermatology,Molecular Biology,Biochemistry

Reference34 articles.

1. Interleukins 1β and 6 but not transforming growth factor-β are essential for the differentiation of interleukin 17-producing human T helper cells;Acosta-Rodriguez;Nat Immunol,2007

2. Surface phenotype and antigenic specificity of human interleukin 17-producing T helper memory cells;Acosta-Rodriguez;Nat Immunol,2007

3. Interleukin-23 promotes a distinct CD4 T cell activation state characterized by the production of interleukin-17;Aggarwal;J Biol Chem,2003

4. TGF-β(1) gene polymorphism in psoriasis vulgaris;Baran;Cytokine,2007

5. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells;Bettelli;Nature,2006

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3. The role of cytokines and vitamin D in vitiligo pathogenesis;Journal of Cosmetic Dermatology;2022-09-05

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5. Cytokines: the yin and yang of vitiligo pathogenesis;Expert Review of Clinical Immunology;2018-11-30

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