An ATP-sensitive potassium channel blocker suppresses sodium-induced hypertension through increased secretion of urinary kallikrein-Reference-Cited by-同舟云学术

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An ATP-sensitive potassium channel blocker suppresses sodium-induced hypertension through increased secretion of urinary kallikrein

Published:2009-03 Issue:3 Volume:32 Page:220-226
ISSN:0916-9636
Container-title:Hypertension Research
language:en
Short-container-title:Hypertens Res

Author:

Kamata Yuji,Fujita Tomoe,Kato Tetsuki,Hayashi Izumi,Kurosaka Maya,Katori Makoto,Fujita Yoshikuni,Majima Masataka

Publisher

Springer Science and Business Media LLC

Subject

Cardiology and Cardiovascular Medicine,Physiology,Internal Medicine

Link

http://www.nature.com/articles/hr200833.pdf

Reference39 articles.

1. Schneider EG, Strandhoy JW, Willis LR, Know FG . Relationship between proximal sodium reabsorption and excretion of calcium, magnesium and phosphate. Kidney Int 1973; 4: 369–376.

2. Margolius HS, Geller R, Pisano JJ, Sjoerdsma A . Altered urinary kallikrein excretion in human hypertension. Lancet 1971; 2: 1063–1065.

3. Margolius HS, Horwitz D, Pisano JJ, Keiser HR . Urinary kallikrein excretion in hypertensive man. Relationships to sodium intake and sodium-retaining steroids. Circ Res 1974; 35: 820–825.

4. Figueroa CD, Maciver AG, Mackenzie JC, Bhoola KD . Localization of immunoreactive kininogen and tissue kallikrein in the human nephron. Histochemistry 1988; 89: 437–442.

5. Katori M, Majima M . The renal kallikrein-kinin system:its role as a safety valve for excess sodium intake, and its attenuation as a possible etiologic factor in solt-sensitive hypertension. Crit Rev Clin Lab Sci 2003; 40: 43–115.

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