Stress hematopoiesis induces a proliferative advantage in TET2 deficiency-Reference-Cited by-同舟云学术

Stress hematopoiesis induces a proliferative advantage in TET2 deficiency

Published:2021-09-30 Issue: Volume: Page:
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Rajan Vinothkumar^ORCID,Collett Keon,Woodside Rachel,Prykhozhij Sergey V.,Moksa Michelle,Carles Annäick,Wong Marcus,Liebman Mira,Hirst Martin^ORCID,Berman Jason N.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

https://www.nature.com/articles/s41375-021-01427-7.pdf

Reference38 articles.

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2. Haferlach T, Nagata Y, Grossmann V, Okuno Y, Bacher U, Nagae G, et al. Landscape of genetic lesions in 944 patients with myelodysplastic syndromes. Leukemia.2013;28:241.

3. Chou W-C, Chou S-C, Liu C-Y, Chen C-Y, Hou H-A, Kuo Y-Y, et al. TET2 mutation is an unfavorable prognostic factor in acute myeloid leukemia patients with intermediate-risk cytogenetics. Blood.2011;118:3803–10.

4. Genovese G, Kähler AK, Handsaker RE, Lindberg J, Rose SA, Bakhoum SF, et al. Clonal hematopoiesis and blood-cancer risk inferred from blood DNA sequence. N. Engl J Med. 2014;371:2477–87.

5. Young AL, Challen GA, Birmann BM, Druley TE. Clonal haematopoiesis harbouring AML-associated mutations is ubiquitous in healthy adults. Nat Commun.2016;7:12484.

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