SETBP1 is dispensable for normal and malignant hematopoiesis-Reference-Cited by-同舟云学术

SETBP1 is dispensable for normal and malignant hematopoiesis

Published:2023-07-18 Issue:9 Volume:37 Page:1802-1811
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Tanaka Atsushi^ORCID,Nishimura Koutarou,Saika Wataru^ORCID,Kon Ayana,Koike Yui,Tatsumi Hiromi,Takeda June^ORCID,Nomura Masaki,Zang Weijia,Nakayama Manabu^ORCID,Matsuda Masashi,Yamazaki Hiromi,Fukumoto Miki,Ito Hiromi,Hayashi Yasutaka,Kitamura Toshio^ORCID,Kawamoto Hiroshi,Takaori-Kondo Akifumi^ORCID,Koseki Haruhiko,Ogawa Seishi,Inoue Daichi^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

https://www.nature.com/articles/s41375-023-01970-5.pdf

Reference48 articles.

1. Cristobal I, Blanco FJ, Garcia-Orti L, Marcotegui N, Vicente C, Rifon J, et al. SETBP1 overexpression is a novel leukemogenic mechanism that predicts adverse outcome in elderly patients with acute myeloid leukemia. Blood. 2010;115:615–25.

2. Makishima H, Yoshida K, Nguyen N, Przychodzen B, Sanada M, Okuno Y, et al. Somatic SETBP1 mutations in myeloid malignancies. Nat Genet. 2013;45:942–6.

3. Shou LH, Cao D, Dong XH, Fang Q, Wu Y, Zhang Y, et al. Prognostic significance of SETBP1 mutations in myelodysplastic syndromes, chronic myelomonocytic leukemia, and chronic neutrophilic leukemia: A meta-analysis. PLoS One. 2017;12:e0171608.

4. Piazza R, Valletta S, Winkelmann N, Redaelli S, Spinelli R, Pirola A, et al. Recurrent SETBP1 mutations in atypical chronic myeloid leukemia. Nat Genet. 2013;45:18–24.

5. Carratt SA, Braun TP, Coblentz C, Schonrock Z, Callahan R, Curtiss BM, et al. Mutant SETBP1 enhances NRAS-driven MAPK pathway activation to promote aggressive leukemia. Leukemia. 2021;35:3594–9.

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