Cryptic genomic lesions in adverse-risk acute myeloid leukemia identified by integrated whole genome and transcriptome sequencing-Reference-Cited by-同舟云学术

Cryptic genomic lesions in adverse-risk acute myeloid leukemia identified by integrated whole genome and transcriptome sequencing

Published:2019-08-21 Issue:1 Volume:34 Page:306-311
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Kim Jaeseung C.,Zuzarte Philip C.,Murphy Tracy,Chan-Seng-Yue Michelle,Brown Andrew M. K.,Krzyzanowski Paul M.^ORCID,Smith Adam C.^ORCID,Notta Faiyaz,Minden Mark D.,McPherson John D.

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/s41375-019-0546-1.pdf

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3. Haferlach C, Dicker F, Herholz H, Schnittger S, Kern W, Haferlach T. Mutations of the TP53 gene in acute myeloid leukemia are strongly associated with a complex aberrant karyotype. Leukemia. 2008;22:1539–41.

4. Welch JS, Westervelt P, Ding L, Larson DE, Klco JM, Kulkarni S, et al. Use of whole-genome sequencing to diagnose a cryptic fusion oncogene. J Am Med Assoc. 2011;305:1577–84.

5. King RL, Naghashpour M, Watt CD, Morrissette JJD, Bagg A. A comparative analysis of molecular genetic and conventional cytogenetic detection of diagnostically important translocations in more than 400 cases of acute leukemia, highlighting the frequency of false-negative conventional cytogenetics. Am J Clin Pathol. 2011;135:921–8.

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