Autocrine Tnf signaling favors malignant cells in myelofibrosis in a Tnfr2-dependent fashion-Reference-Cited by-同舟云学术

Autocrine Tnf signaling favors malignant cells in myelofibrosis in a Tnfr2-dependent fashion

Published:2018-04-18 Issue:11 Volume:32 Page:2399-2411
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Heaton William L.,Senina Anna V.,Pomicter Anthony D.,Salama Mohamed E.,Clair Phillip M.,Yan Dongqing,Bell Russell N.,Gililland Jeremy M.,Prchal Josef T.,O’Hare Thomas,Deininger Michael W.

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/s41375-018-0131-z.pdf

Reference57 articles.

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2. Fleischman AG, Aichberger KJ, Luty SB, Bumm TG, Petersen CL, Doratotaj S, et al. TNFalpha facilitates clonal expansion of JAK2V617F positive cells in myeloproliferative neoplasms. Blood. 2011;118:6392–8.

3. James C, Ugo V, Le Couedic J-P, Staerk J, Delhommeau F, Lacout C, et al. A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera. Nature. 2005;434:1144–8.

4. Pikman Y, Lee BH, Mercher T, McDowell E, Ebert BL, Gozo M, et al. MPLW515L is a novel somatic activating mutation in myelofibrosis with myeloid metaplasia. PLoS Med. 2006;3:e270.

5. Nangalia J, Massie CE, Baxter EJ, Nice FL, Gundem G, Wedge DC, et al. Somatic CALR mutations in myeloproliferative neoplasms with nonmutated JAK2. New Engl J Med. 2013;369:2391–405.

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