Lack of association between STK39 and hypertension in the Chinese population
Author:
Publisher
Springer Science and Business Media LLC
Subject
Internal Medicine
Link
http://www.nature.com/articles/jhh201246.pdf
Reference21 articles.
1. Wang Y, O'Connell JR, McArdle PF, Wade JB, Dorff SE, Shah SJ et al. Whole-genome association study identifies STK39 as a hypertension susceptibility gene. Proc Natl Acad Sci USA 2009; 106 (1): 226–231.
2. Fava C, Danese E, Montagnana M, Sjogren M, Almgren P, Engstrom G et al. Serine/threonine kinase 39 is a candidate gene for primary hypertension especially in women: results from two cohort studies in Swedes. J Hypertens 2011; 29 (3): 484–491.
3. Vitari AC, Deak M, Morrice NA, Alessi DR . The WNK1 and WNK4 protein kinases that are mutated in Gordon's hypertension syndrome phosphorylate and activate SPAK and OSR1 protein kinases. Biochem J 2005; 391 (Pt 1): 17–24.
4. Moriguchi T, Urushiyama S, Hisamoto N, Iemura S, Uchida S, Natsume T et al. WNK1 regulates phosphorylation of cation-chloride-coupled cotransporters via the STE20-related kinases, SPAK and OSR1. J Biol Chem 2005; 280 (52): 42685–42693.
5. Delpire E, Gagnon KB . SPAK and OSR1: STE20 kinases involved in the regulation of ion homoeostasis and volume control in mammalian cells. Biochem J 2008; 409 (2): 321–331.
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