Push back to respond better: regulatory inhibition of the DNA double-strand break response
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Molecular Biology
Link
http://www.nature.com/articles/nrm3659.pdf
Reference119 articles.
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3. Chowdhury, D. et al. γ-H2AX dephosphorylation by protein phosphatase 2A facilitates DNA double-strand break repair. Mol. Cell 20, 801–809 (2005).
4. Gudjonsson, T. et al. TRIP12 and UBR5 suppress spreading of chromatin ubiquitylation at damaged chromosomes. Cell 150, 697–709 (2012). Demonstrates that RNF168 is a limiting component of the DSB response and that the pool of RNF168 molecules is kept in check by the E3 ubiquitin ligases TRIP12 and UBR5.
5. Nakada, S., Chen, G. I., Gingras, A. C. & Durocher, D. PP4 is a γH2AX phosphatase required for recovery from the DNA damage checkpoint. EMBO Rep. 9, 1019–1026 (2008).
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