The Unc93b1 mutation 3d disrupts exogenous antigen presentation and signaling via Toll-like receptors 3, 7 and 9-Reference-Cited by-同舟云学术

The Unc93b1 mutation 3d disrupts exogenous antigen presentation and signaling via Toll-like receptors 3, 7 and 9

Published:2006-01-15 Issue:2 Volume:7 Page:156-164
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Tabeta Koichi,Hoebe Kasper,Janssen Edith M,Du Xin,Georgel Philippe,Crozat Karine,Mudd Suzanne,Mann Navjiwan,Sovath Sosathya,Goode Jason,Shamel Louis,Herskovits Anat A,Portnoy Daniel A,Cooke Michael,Tarantino Lisa M,Wiltshire Tim,Steinberg Benjamin E,Grinstein Sergio,Beutler Bruce

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/ni1297.pdf

Reference55 articles.

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3. Takeuchi, O. et al. Discrimination of bacterial lipoproteins by Toll-like receptor 6. Int. Immunol. 13, 933–940 (2001).

4. Gantner, B.N., Simmons, R.M., Canavera, S.J., Akira, S. & Underhill, D.M. Collaborative induction of inflammatory responses by dectin-1 and Toll-like receptor 2. J. Exp. Med. 197, 1107–1117 (2003).

5. Hayashi, F. et al. The innate immune response to bacterial flagellin is mediated by Toll- like receptor 5. Nature 410, 1099–1103 (2001).

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