Predominance and high antibiotic resistance of the emergingClostridium difficilegenotypes NAPCR1and NAP9 in a Costa Rican hospital over a 2-year period without outbreaks-Reference-Cited by-同舟云学术

Predominance and high antibiotic resistance of the emergingClostridium difficilegenotypes NAPCR1and NAP9 in a Costa Rican hospital over a 2-year period without outbreaks

Published:2016-01 Issue:1 Volume:5 Page:1-5
ISSN:2222-1751
Container-title:Emerging Microbes & Infections
language:en
Short-container-title:Emerging Microbes & Infections

Author:

López-Ureña Diana¹,Quesada-Gómez Carlos¹,Montoya-Ramírez Mónica¹,del Mar Gamboa-Coronado María¹,Somogyi Teresita¹,Rodríguez César¹,Rodríguez-Cavallini Evelyn¹

Affiliation:

1. Laboratory of Research in Anaerobic Bacteriology and Research Center in Tropical Diseases, University of Costa Rica, San José 11501-2060, Costa Rica

Publisher

Informa UK Limited

Subject

Virology,Infectious Diseases,Drug Discovery,General Medicine,Immunology,Microbiology,Parasitology,Epidemiology

Link

http://www.nature.com/articles/emi201638.pdf

Reference46 articles.

1. Antibiotics and hospital-acquired Clostridium difficile infection: update of systematic review and meta-analysis

2. Clinical Factors Associated With Development of Severe-Complicated Clostridium difficile Infection

3. Bacterial protein toxins that modify host regulatory GTPases

4. Clostridium difficile Toxin A–Induced Colonocyte Apoptosis Involves p53-Dependent p21(WAF1/CIP1) Induction via p38 Mitogen-Activated Protein Kinase

5. Clostridium difficile Toxin B Causes Epithelial Cell Necrosis through an Autoprocessing-Independent Mechanism

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