Acquired radioresistance of human tumor cells by DNA-PK/AKT/GSK3β-mediated cyclin D1 overexpression-Reference-Cited by-同舟云学术

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Acquired radioresistance of human tumor cells by DNA-PK/AKT/GSK3β-mediated cyclin D1 overexpression

Published:2010-06-21 Issue:34 Volume:29 Page:4826-4837
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Shimura T,Kakuda S,Ochiai Y,Nakagawa H,Kuwahara Y,Takai Y,Kobayashi J,Komatsu K,Fukumoto M

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2010238.pdf

Reference48 articles.

1. Agami R, Bernards R . (2000). Distinct initiation and maintenance mechanisms cooperate to induce G1 cell cycle arrest in response to DNA damage. Cell 102: 55–66.

2. Alao JP . (2007). The regulation of cyclin D1 degradation: roles in cancer development and the potential for therapeutic invention. Mol Cancer 6: 24.

3. Alt JR, Cleveland JL, Hannink M, Diehl JA . (2000). Phosphorylation-dependent regulation of cyclin D1 nuclear export and cyclin D1-dependent cellular transformation. Genes Dev 14: 3102–3114.

4. Arnaudeau C, Lundin C, Helleday T . (2001). DNA double-strand breaks associated with replication forks are predominantly repaired by homologous recombination involving an exchange mechanism in mammalian cells. J Mol Biol 307: 1235–1245.

5. Baldin V, Lukas J, Marcote MJ, Pagano M, Draetta G . (1993). Cyclin D1 is a nuclear protein required for cell cycle progression in G1. Genes Dev 7: 812–821.

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