The H3K27me3 demethylase, KDM6B, is induced by Epstein–Barr virus and over-expressed in Hodgkin's Lymphoma-Reference-Cited by-同舟云学术

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The H3K27me3 demethylase, KDM6B, is induced by Epstein–Barr virus and over-expressed in Hodgkin's Lymphoma

Published:2011-01-17 Issue:17 Volume:30 Page:2037-2043
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Anderton J A,Bose S,Vockerodt M,Vrzalikova K,Wei W,Kuo M,Helin K,Christensen J,Rowe M,Murray P G,Woodman C B

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2010579.pdf

Reference32 articles.

1. Agger K, Cloos PA, Rudkjær L, Williams K, Andersen G, Christensen J et al. (2009). The H3K27me3 demethylase JMJD3 contributes to the activation of the INK4A – ARF locus in response to oncogene- and stress-induced senescence. Genes Dev 23: 1171–1176.

2. Agger K, Cloos Pa, Christensen J, Pasini D, Rose S, Rappsilber J et al. (2007). UTX and JMJD3 are histone H3K27 demethylases involved in HOX gene regulation and development. Nature 449: 731–734.

3. Agherbi H, Gaussmann-Wenger A, Verthuy C, Chasson L, Serrano M, Djabali M et al. (2009). Polycomb mediated epigenetic silencing and replication timing at the INK4a/ARF locus during senescence. PloS one 4: e5622. doi: 10.1371/journal.pone.0005622.

4. Aldinucci D, Gloghini A, Pinto A, De Filippi R, Carbone A . (2010). The classical Hodgkin's lymphoma microenvironment and its role in promoting tumour growth and immune escape. J Pathol 221: 248–263.

5. Barradas M, Anderton E, Acosta JC, Li S, Banito A, Rodriguez-Niedenführ M et al. (2009). Histone demethylase JMJD3 contributes to epigenetic control of INK4a/ARF by oncogenic RAS. Genes Dev 23: 1177–1182.

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