Genome-wide shRNA screen reveals increased mitochondrial dependence upon mTORC2 addiction-Reference-Cited by-同舟云学术

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Genome-wide shRNA screen reveals increased mitochondrial dependence upon mTORC2 addiction

Published:2010-12-20 Issue:13 Volume:30 Page:1551-1565
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Colombi M,Molle K D,Benjamin D,Rattenbacher-Kiser K,Schaefer C,Betz C,Thiemeyer A,Regenass U,Hall M N,Moroni C

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2010539.pdf

Reference46 articles.

1. Alto NM, Soderling J, Scott JD . (2002). Rab32 is an A-kinase anchoring protein and participates in mitochondrial dynamics. J Cell Biol 158: 659–668.

2. Bandyopadhyay G, Sajan MP, Kanoh Y, Standaert ML, Quon MJ, Reed BC et al. (2001). Glucose activates protein kinase C-zeta/lambda through proline-rich tyrosine kinase-2, extracellular signal-regulated kinase, and phospholipase D: a novel mechanism for activating glucose transporter translocation. J Biol Chem 276: 35537–35545.

3. Bauer DE, Harris MH, Plas DR, Lum JJ, Hammerman PS, Rathmell JC et al. (2004). Cytokine stimulation of aerobic glycolysis in hematopoietic cells exceeds proliferative demand. FASEB J 18: 1303–1305.

4. Bentzinger CF, Romanino K, Cloetta D, Lin S, Mascarenhas JB, Oliveri F et al. (2008). Skeletal muscle-specific ablation of raptor, but not of rictor, causes metabolic changes and results in muscle dystrophy. Cell Metab 8: 411–424.

5. Chiang GG, Abraham RT . (2007). Targeting the mTOR signaling network in cancer. Trends Mol Med 13: 433–442.

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