Human Tra2 proteins jointly control a CHEK1 splicing switch among alternative and constitutive target exons-Reference-Cited by-同舟云学术

Human Tra2 proteins jointly control a CHEK1 splicing switch among alternative and constitutive target exons

Published:2014-09-11 Issue:1 Volume:5 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Best Andrew,James Katherine,Dalgliesh Caroline,Hong Elaine,Kheirolahi-Kouhestani Mahsa,Curk Tomaz,Xu Yaobo,Danilenko Marina,Hussain Rafiq,Keavney Bernard,Wipat Anil,Klinck Roscoe,Cowell Ian G.,Cheong Lee Ka,Austin Caroline A.,Venables Julian P.,Chabot Benoit,Santibanez Koref Mauro,Tyson-Capper Alison,Elliott David J.

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms5760.pdf

Reference74 articles.

1. Fox-Walsh, K. L. & Hertel, K. J. Splice-site pairing is an intrinsically high fidelity process. Proc. Natl Acad. Sci. USA 106, 1766–1771 (2009).

2. Kelemen, O. et al. Function of alternative splicing. Gene 514, 1–30 (2013).

3. Djebali, S. et al. Landscape of transcription in human cells. Nature 489, 101–108 (2012).

4. Ehrmann, I. et al. The tissue-specific RNA binding protein T-STAR controls regional splicing patterns of neurexin pre-mRNAs in the brain. PLoS Genet 9, e1003474 (2013).

5. Venables, J. P. et al. RBFOX2 is an important regulator of mesenchymal tissue-specific splicing in both normal and cancer tissues. Mol. Cell Biol. 33, 396–405 (2013).

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