The phasor-FLIM fingerprints reveal shifts from OXPHOS to enhanced glycolysis in Huntington Disease
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/srep34755.pdf
Reference43 articles.
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2. Heikal, A. A. Intracellular coenzymes as natural biomarkers for metabolic activities and mitochondrial anomalies. Biomarkers in medicine 4, 241–263 (2010).
3. Podolsky, S., Leopold, N. & Sax, D. Increased frequency of diabetes mellitus in patients with Huntington’s chorea. The Lancet 299, 1356–1359 (1972).
4. Hurlbert, M. S. et al. Mice transgenic for an expanded CAG repeat in the Huntington’s disease gene develop diabetes. DIABETES 48, 649–651, doi: 10.2337/diabetes.48.3.649 (1999).
5. Oláh, J. et al. Increased glucose metabolism and ATP level in brain tissue of Huntington’s disease transgenic mice. FEBS Journal 275, 4740–4755, doi: 10.1111/j.1742-4658.2008.06612.x (2008).
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