p16Ink4a-induced senescence of pancreatic beta cells enhances insulin secretion-Reference-Cited by-同舟云学术

p16Ink4a-induced senescence of pancreatic beta cells enhances insulin secretion

Published:2016-03-07 Issue:4 Volume:22 Page:412-420
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Helman Aharon,Klochendler Agnes,Azazmeh Narmen,Gabai Yael,Horwitz Elad,Anzi Shira,Swisa Avital,Condiotti Reba,Granit Roy Z,Nevo Yuval,Fixler Yaakov,Shreibman Dorin,Zamir Amit,Tornovsky-Babeay Sharona,Dai Chunhua,Glaser Benjamin,Powers Alvin C,Shapiro A M James,Magnuson Mark A,Dor Yuval,Ben-Porath Ittai

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm.4054.pdf

Reference66 articles.

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3. Storer, M. et al. Senescence is a developmental mechanism that contributes to embryonic growth and patterning. Cell 155, 1119–1130 (2013).

4. Muñoz-Espín, D. et al. Programmed cell senescence during mammalian embryonic development. Cell 155, 1104–1118 (2013).

5. Kim, W.Y. & Sharpless, N.E. The regulation of INK4-ARF in cancer and aging. Cell 127, 265–275 (2006).

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