Tyrosine kinases as targets for the treatment of rheumatoid arthritis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Rheumatology
Link
http://www.nature.com/articles/nrrheum.2009.82.pdf
Reference80 articles.
1. Firestein, G. S. Evolving concepts of rheumatoid arthritis. Nature 423, 356–361 (2003).
2. Sano, H. et al. Coexpression of phosphotyrosine-containing proteins, platelet-derived growth factor-B, and fibroblast growth factor-1 in situ in synovial tissues of patients with rheumatoid arthritis and Lewis rats with adjuvant or streptococcal cell wall arthritis. J. Clin. Invest. 91, 553–565 (1993).
3. Schlessinger, J. Cell signaling by receptor tyrosine kinases. Cell 103, 211–225 (2000).
4. Benati, D. & Baldari, C. T. SRC family kinases as potential therapeutic targets for malignancies and immunological disorders. Curr. Med. Chem. 15, 1154–1165 (2008).
5. Schindler, T. et al. Structural mechanism for STI-571 inhibition of Abelson tyrosine kinase. Science 289, 1938–1942 (2000).
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