Rosiglitazone attenuates myocardial remodeling in spontaneously hypertensive rats-Reference-Cited by-同舟云学术

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Rosiglitazone attenuates myocardial remodeling in spontaneously hypertensive rats

Published:2011-01-20 Issue:3 Volume:34 Page:354-360
ISSN:0916-9636
Container-title:Hypertension Research
language:en
Short-container-title:Hypertens Res

Author:

Ti Yun,Hao Ming-Xiu,Li Chuan-Bao,Wang Zhi-Hao,Hou Xiao-Yang,Zhao Xue-Qiang,Liu Jun-Ni,Zhang Wei,Zhang Yun,Bu Pei-Li

Publisher

Springer Science and Business Media LLC

Subject

Cardiology and Cardiovascular Medicine,Physiology,Internal Medicine

Link

http://www.nature.com/articles/hr2010242.pdf

Reference26 articles.

1. Spinale FG . Myocardial matrix remodeling and the matrix metalloproteinases: influence on cardiac form and function. Physiol Rev 2007; 87: 1285–1342.

2. Deng T, Karin M . c-Fos transcriptional activity stimulated by H-Ras-activated protein kinase distinct from JNK and ERK. Nature 1994; 371: 171–175.

3. Li X, Meng Y, Wu P, Zhang Z, Yang X . Angiotensin II and aldosterone stimulating NF-kappaB and AP-1 activation in hepatic fibrosis of rat. Regul Pept 2007; 138: 15–25.

4. Yamamoto K, Ohki R, Lee RT, Ikeda U, Shimada K . Peroxisome proliferator-activated receptor gamma activators inhibit cardiac hypertrophy in cardiomyocytes. Circulation 2001; 104: 1670–1675.

5. Ren L, Li Y, Li Y, Tang R, Hu D, Sheng Z, Liu N . Inhibitory effects of rosiglitazone on cardiac hypertrophy through modulating the renin-angiotensin system in diet-induced hypercholesterolemic rats. Cell Biochem Funct 2010; 28: 58–65.

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The CD147/MMP-2 signaling pathway may regulate early stage cardiac remodelling in spontaneously hypertensive rats;Clinical and Experimental Pharmacology and Physiology;2016-10-20

2. Protective effects of pioglitazone against immunoglobulin deposition on heart of streptozotocin-induced diabetic rats;Journal of Endocrinological Investigation;2014-01-09

3. NOD2 Deficiency Protects against Cardiac Remodeling after Myocardial Infarction in Mice;Cellular Physiology and Biochemistry;2013

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