mTORC1 inhibitor rapamycin and ER stressor tunicamycin induce differential patterns of ER-mitochondria coupling-Reference-Cited by-同舟云学术

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mTORC1 inhibitor rapamycin and ER stressor tunicamycin induce differential patterns of ER-mitochondria coupling

Published:2016-11-03 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Bravo-Sagua Roberto,López-Crisosto Camila,Parra Valentina,Rodriguez-Peña Marcelo,Rothermel Beverly A.,Quest Andrew F.G.,Lavandero Sergio

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep36394.pdf

Reference46 articles.

1. Bravo, R. et al. Endoplasmic reticulum: ER stress regulates mitochondrial bioenergetics. Int J Biochem Cell Biol 44, 16–20 (2012).

2. Bravo-Sagua, R. et al. Cell death and survival through the endoplasmic reticulum- mitochondrial axis. Curr Mol Med 13, 317–329 (2013).

3. Bravo-Sagua, R. et al. Organelle communication: signaling crossroads between homeostasis and disease. Int J Biochem Cell Biol 50, 55–59 (2014).

4. Simmen, T., Lynes, E. M., Gesson, K. & Thomas, G. Oxidative protein folding in the endoplasmic reticulum: tight links to the mitochondria-associated membrane (MAM). Biochim Biophys Acta 1798, 1465–1473 (2010).

5. Cárdenas, C. et al. Essential regulation of cell bioenergetics by constitutive InsP3 receptor Ca2+ transfer to mitochondria. Cell 142, 270–283 (2010).

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