Clonal heterogeneity of acute myeloid leukemia treated with the IDH2 inhibitor enasidenib-Reference-Cited by-同舟云学术

Clonal heterogeneity of acute myeloid leukemia treated with the IDH2 inhibitor enasidenib

Published:2018-07-16 Issue:8 Volume:24 Page:1167-1177
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Quek Lynn,David Muriel D.^ORCID,Kennedy Alison,Metzner Marlen,Amatangelo Michael,Shih Alan,Stoilova Bilyana^ORCID,Quivoron Cyril^ORCID,Heiblig Maël^ORCID,Willekens Christophe^ORCID,Saada Véronique^ORCID,Alsafadi Samar,Vijayabaskar M. S.,Peniket Andy,Bernard Oliver A.,Agresta Sam,Yen Katharine,MacBeth Kyle,Stein Eytan,Vassiliou George S.^ORCID,Levine Ross,De Botton Stephane,Thakurta Anjan,Penard-Lacronique Virginie^ORCID,Vyas Paresh^ORCID

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/s41591-018-0115-6.pdf

Reference56 articles.

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3. Cancer Genome Atlas Research et al. Genomic and epigenomic landscapes of adult de novo acute myeloid leukemia. N. Engl. J. Med. 368, 2059–2074 (2013).

4. Paschka, P. et al. IDH1 and IDH2 mutations are frequent genetic alterations in acute myeloid leukemia and confer adverse prognosis in cytogenetically normal acute myeloid leukemia with NPM1 mutation without FLT3 internal tandem duplication. J. Clin. Oncol. 28, 3636–3643 (2010).

5. Green, C. L. et al. The prognostic significance of IDH2 mutations in AML depends on the location of the mutation. Blood 118, 409–412 (2011).

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