Protection against doxorubicin-induced myocardial dysfunction in mice by cardiac-specific expression of carboxyl terminus of hsp70-interacting protein-Reference-Cited by-同舟云学术

Protection against doxorubicin-induced myocardial dysfunction in mice by cardiac-specific expression of carboxyl terminus of hsp70-interacting protein

Published:2016-06 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Wang Lei,Zhang Tian-Peng,Zhang Yuan,Bi Hai-Lian,Guan Xu-Min,Wang Hong-Xia,Wang Xia,Du Jie,Xia Yun-Long,Li Hui-Hua

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep28399.pdf

Reference55 articles.

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2. Lotrionte, M. et al. Review and meta-analysis of incidence and clinical predictors of anthracycline cardiotoxicity. The American journal of cardiology 112, 1980–1984 (2013).

3. Singal, P. K., Li, T., Kumar, D., Danelisen, I. & Iliskovic, N. Adriamycin-induced heart failure: mechanism and modulation. Molecular and cellular biochemistry 207, 77–86 (2000).

4. Kim, Y. K. et al. Deletion of the inducible 70-kDa heat shock protein genes in mice impairs cardiac contractile function and calcium handling associated with hypertrophy. Circulation 113, 2589–2597 (2006).

5. Fan, G. C. et al. Heat shock protein 20 interacting with phosphorylated Akt reduces doxorubicin-triggered oxidative stress and cardiotoxicity. Circulation research 103, 1270–1279 (2008).

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