Aberrantly glycosylated IgA1 induces mesangial cells to produce platelet-activating factor that mediates nephrin loss in cultured podocytes
Author:
Publisher
Elsevier BV
Subject
Nephrology
Reference39 articles.
1. Aberrant glycosylation in IgA nephropathy (IgAN);Coppo;Kidney Int,2004
2. Galactosylation of N-and O-linked carbohydrate moieties of IgA1 and IgG in IgA nephropathy;Allen;Clin Exp Immunol,1995
3. Galactose-deficient IgA1 in sera of IgA nephropathy patients is present in complexes with IgG;Tomana;Kidney Int,1997
4. Analyses of IgA1 hinge glycopeptides in IgA nephropathy by matrix-assisted laser desorption/ionization time-of-flight mass spectrometry;Hiki;J Am Soc Nephrol,1998
5. Aberrantly glycosylated IgA molecules downregulate the synthesis and secretion of vascular endothelial growth factor in human mesangial cells;Amore;Am J Kidney Dis,2000
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