Genetic deletion of the angiotensin-(1–7) receptor Mas leads to glomerular hyperfiltration and microalbuminuria-Reference-Cited by-同舟云学术

Genetic deletion of the angiotensin-(1–7) receptor Mas leads to glomerular hyperfiltration and microalbuminuria

Published:2009-06 Issue:11 Volume:75 Page:1184-1193
ISSN:0085-2538
Container-title:Kidney International
language:en
Short-container-title:Kidney International

Author:

Pinheiro Sérgio V.B.,Ferreira Anderson J.,Kitten Gregory T.,da Silveira Kátia D.,da Silva Deivid A.,Santos Sérgio H.S.,Gava Elisandra,Castro Carlos H.,Magalhães Júnio A.,da Mota Renata K.,Botelho-Santos Giancarla A.,Bader Michael,Alenina Natalia,Santos Robson A.S.,Simoes e Silva Ana Cristina

Publisher

Elsevier BV

Subject

Nephrology

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4. Recent advances in the angiotensin-converting enzyme 2-angiotensin(1–7)-Mas axis;Santos;Exp Physiol,2008

5. Evidence that the angiotensin IV (AT(4)) receptor is the enzyme insulin-regulated aminopeptidase;Albiston;J Biol Chem,2001

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