Author:
Tardito Saverio,Oudin Anaïs,Ahmed Shafiq U.,Fack Fred,Keunen Olivier,Zheng Liang,Miletic Hrvoje,Sakariassen Per Øystein,Weinstock Adam,Wagner Allon,Lindsay Susan L.,Hock Andreas K.,Barnett Susan C.,Ruppin Eytan,Mørkve Svein Harald,Lund-Johansen Morten,Chalmers Anthony J.,Bjerkvig Rolf,Niclou Simone P.,Gottlieb Eyal
Publisher
Springer Science and Business Media LLC
Reference42 articles.
1. Moreadith, R. W. & Lehninger, A. L. The pathways of glutamate and glutamine oxidation by tumor cell mitochondria. Role of mitochondrial NAD(P) + -dependent malic enzyme. J. Biol. Chem. 259, 6215–6221 (1984).
2. Yuneva, M. O. et al. The metabolic profile of tumors depends on both the responsible genetic lesion and tissue type. Cell Metab. 15, 157–170 (2012).
3. Wise, D. R. et al. Myc regulates a transcriptional program that stimulates mitochondrial glutaminolysis and leads to glutamine addiction. Proc. Natl Acad. Sci. USA 105, 18782–18787 (2008).
4. DeBerardinis, R. J. et al. Beyond aerobic glycolysis: transformed cells can engage in glutamine metabolism that exceeds the requirement for protein and nucleotide synthesis. Proc. Natl Acad. Sci. USA 104, 19345–19350 (2007).
5. Tardito, S. et al. L-Asparaginase and inhibitors of glutamine synthetase disclose glutamine addiction of β-catenin-mutated human hepatocellular carcinoma cells. Curr. Cancer Drug Targets 11, 929–943 (2011).
Cited by
409 articles.
订阅此论文施引文献
订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献