Glucocorticoids suppress inflammation via the upregulation of negative regulator IRAK-M-Reference-Cited by-同舟云学术

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Glucocorticoids suppress inflammation via the upregulation of negative regulator IRAK-M

Published:2015-01-14 Issue:1 Volume:6 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Miyata Masanori,Lee Ji-Yun,Susuki-Miyata Seiko,Wang Wenzhuo Y.,Xu Haidong,Kai Hirofumi,Kobayashi Koichi S.,Flavell Richard A.,Li Jian-Dong

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms7062.pdf

Reference64 articles.

1. Quax, R. A. et al. Glucocorticoid sensitivity in health and disease. Nat. Rev. Endocrinol. 9, 670–686 (2013).

2. Clark, A. R. & Belvisi, M. G. Maps and legends: the quest for dissociated ligands of the glucocorticoid receptor. Pharmacol. Ther. 134, 54–67 (2012).

3. Beck, I. M. et al. Crosstalk in inflammation: the interplay of glucocorticoid receptor-based mechanisms and kinases and phosphatases. Endocr. Rev. 30, 830–882 (2009).

4. Luecke, H. F. & Yamamoto, K. R. The glucocorticoid receptor blocks P-TEFb recruitment by NFkappaB to effect promoter-specific transcriptional repression. Genes Dev. 19, 1116–1127 (2005).

5. Busillo, J. M. & Cidlowski, J. A. The five Rs of glucocorticoid action during inflammation: ready, reinforce, repress, resolve, and restore. Trends Endocrinol. Metab. 24, 109–119 (2013).

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