C/EBPα mutations in acute myeloid leukaemias
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Earth and Planetary Sciences,General Environmental Science
Link
http://www.nature.com/articles/nrc1363.pdf
Reference57 articles.
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2. Wang, N. D. et al. Impaired energy homeostasis in C/EBPα knockout mice. Science 269, 1108–1112 (1995). This paper demonstrates the requirement for C/EBPα during white-fat adipogenesis.
3. Zhang, D. E. et al. Absence of granulocyte colony-stimulating factor signaling and neutrophil development in CCAAT enhancer binding protein α-deficient mice. Proc. Natl Acad. Sci. USA 94, 569–574 (1997). This paper provided the first demonstration of a block in granulopoiesis in the absence of the C/EBPα protein.
4. Freytag, S. O., Paielli, D. L. & Gilbert, J. D. Ectopic expression of the CCAAT/enhancer-binding protein α promotes the adipogenic program in a variety of mouse fibroblastic cells. Genes Dev. 8, 1654–1663 (1994).
5. Radomska, H. S. et al. CCAAT/enhancer binding protein α is a regulatory switch sufficient for induction of granulocytic development from bipotential myeloid progenitors. Mol. Cell. Biol. 18, 4301–4314 (1998).
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