Deficiency of interleukin-18 in mice leads to hyperphagia, obesity and insulin resistance-Reference-Cited by-同舟云学术

Deficiency of interleukin-18 in mice leads to hyperphagia, obesity and insulin resistance

Published:2006-05-28 Issue:6 Volume:12 Page:650-656
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Netea Mihai G,Joosten Leo A B,Lewis Eli,Jensen Dalan R,Voshol Peter J,Kullberg Bart Jan,Tack Cees J,van Krieken Han,Kim Soo-Hyun,Stalenhoef Anton F,van de Loo Fons A,Verschueren Ineke,Pulawa Leslie,Akira Shizuo,Eckel Robert H,Dinarello Charles A,van den Berg Wim,van der Meer Jos W M

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm1415.pdf

Reference25 articles.

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2. Netea, M.G. et al. Neutralization of interleukin-18 reduces neutrophil tissue accumulation and protects mice against lethal Escherichia coli and Salmonella typhimurium endotoxemia. J. Immunol. 164, 2644–2649 (2000).

3. Dinarello, C.A. IL-18: A Th1-inducing, proinflammatory cytokine and a new member of the IL-1 family. J. Allergy Clin. Immunol. 103, 11–24 (1999).

4. Ghayur, T. et al. Caspase-1 processes IFN-γ-inducing factor and regulates LPS-induced IFN-γ production. Nature 386, 619–623 (1997).

5. Novick, D. et al. Interleukin-18 binding protein: a novel modulator of the Th1 cytokine response. Immunity 10, 127–136 (1999).

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