NOS: Modulator, not mediator of cardiac performance
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
http://www.nature.com/articles/nm0399_273.pdf
Reference19 articles.
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2. Balligand, J.-L. et al. Nitric oxide-dependent parasympathetic signaling is due to activation of constitutive endothelial (type III) nitric oxide synthase in cardiac myocytes. J. Biol. Chem. 270, 14582 –14586 (1995).
3. Feron, O. et al. Modulation of endothelial nitric-oxide synthase-caveolin interaction in cardiac myocytes. J. Biol. Chem. 273, 30249–30254 (1998).
4. Bates, T.E., Loesch, A., Burnstock, G. & Clark, J.B. Mitochondrial nitric oxide synthase: A ubiquitous regulator of oxidative phosphorylation? Biochem. Biophys. Res. Comm. 218, 40– 44 (1996).
5. Balligand, J.-L. et al. Cytokine-inducible nitric-oxide synthase (iNOS) expression in cardiac myocytes: Characterization and regulation of iNOS expression and detection of iNOS activity in single cardiac myocytes in vitro. J. Biol. Chem. 269, 27580–27588 (1994).
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