PERK silence inhibits glioma cell growth under low glucose stress by blockage of p-AKT and subsequent HK2's mitochondria translocation-Reference-Cited by-同舟云学术

PERK silence inhibits glioma cell growth under low glucose stress by blockage of p-AKT and subsequent HK2's mitochondria translocation

Published:2015-03-12 Issue:1 Volume:5 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Hou Xu,Liu Yaohua,Liu Huailei,Chen Xin,Liu Min,Che Hui,Guo Fei,Wang Chunlei,Zhang Daming,Wu Jianing,Chen Xiaofeng,Shen Chen,Li Chenguang,Peng Fei,Bi Yunke,Yang Zhuowen,Yang Guang,Ai Jing,Gao Xin,Zhao Shiguang

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep09065.pdf

Reference39 articles.

1. Kathagen, A. et al. Hypoxia and oxygenation induce a metabolic switch between pentose phosphate pathway and glycolysis in glioma stem-like cells. Acta Neuropathol. 126, 763–80 (2013).

2. Hanahan, D. & Weinberg, R. A. Hallmarks of cancer: the next generation. Cell 144, 646–74 (2011).

3. Lai, J. H. et al. Nodal regulates energy metabolism in glioma cells by inducing expression of hypoxia-inducible factor 1alpha. Neuro Oncol. 15, 1330–41 (2013).

4. Sasaki, M. et al. IDH1 (R132H) mutation increases murine haematopoietic progenitors and alters epigenetics. Nature 488, 656–9 (2012).

5. Wolf, A. et al. Hexokinase 2 is a key mediator of aerobic glycolysis and promotes tumor growth in human glioblastoma multiforme. J Exp Med. 208, 313–26 (2011).

Cited by 66 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The unfolded protein response machinery in glioblastoma genesis, chemoresistance and as a druggable target;CNS Neuroscience & Therapeutics;2024-07

2. Glucose-driven histone lactylation promotes the immunosuppressive activity of monocyte-derived macrophages in glioblastoma;Immunity;2024-05

3. DNA Damage-Induced Apoptosis Suppressor Triggers Progression and Stemness of Glioma by Enhancing Lymphoid Enhancer-Binding Factor 1 Expression;World Journal of Oncology;2024-04

4. Heparanase interacting BCLAF1 to promote the development and drug resistance of ICC through the PERK/eIF2α pathway;Cancer Gene Therapy;2024-03-11

5. Polo-like kinase 4 promotes tumorigenesis and glucose metabolism in glioma by activating AKT1 signaling;Cancer Letters;2024-03