MSK1 activation in acute myeloid leukemia cells with FLT3 mutations
Author:
Publisher
Springer Science and Business Media LLC
Subject
Oncology,Cancer Research,Hematology
Link
http://www.nature.com/articles/leu201048.pdf
Reference8 articles.
1. Wadleigh M, DeAngelo DJ, Griffin JD, Stone RM . After chronic myelogenous leukemia: tyrosine kinase inhibitors in other hematologic malignancies. Blood 2005; 105: 22–30.
2. Stone RM, DeAngelo DJ, Klimek V, Galinsky I, Estey E, Nimer SD et al. Patients with acute myeloid leukemia and an activating mutation in FLT3 respond to a small-molecule FLT3 tyrosine kinase inhibitor, PKC412. Blood 2005; 105: 54–60.
3. Thomson S, Clayton AL, Hazzalin CA, Rose S, Barratt MJ, Mahadevan LC . The nucleosomal response associated with immediate-early gene induction is mediated via alternative MAP kinase cascades: MSK1 as a potential histone H3/HMG-14 kinase. EMBO J 1999; 18: 4779–4793.
4. Furukawa Y, Vu HA, Akutsu M, Odgerel T, Izumi T, Tsunoda S et al. Divergent cytotoxic effects of PKC412 in combination with conventional antileukemic agents in FLT3 mutation-positive versus negative leukemia cell lines. Leukemia 2007; 21: 1005–1014.
5. Kim H-G, Lee KW, Cho Y-Y, Kang NJ, Oh S-M, Bode AM et al. Mitogen- and stress-activated kinase 1-mediated histone H3 phosphorylation is crucial for cell transformation. Cancer Res 2008; 68: 2538–2547.
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