Correction of B-cell development in Btk-deficient mice using lentiviral vectors with codon-optimized human BTK-Reference-Cited by-同舟云学术

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Correction of B-cell development in Btk-deficient mice using lentiviral vectors with codon-optimized human BTK

Published:2010-06-24 Issue:9 Volume:24 Page:1617-1630
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Ng Y Y,Baert M R M,Pike-Overzet K,Rodijk M,Brugman M H,Schambach A,Baum C,Hendriks R W,van Dongen J J M,Staal F J T

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/leu2010140.pdf

Reference58 articles.

1. Fischer A . Human primary immunodeficiency diseases: a perspective. Nat Immunol 2004; 5: 23–30.

2. Cavazzana-Calvo M, Fischer A . Gene therapy for severe combined immunodeficiency: are we there yet? J Clin Invest 2007; 117: 1456–1465.

3. Pike-Overzet K, van der Burg M, Wagemaker G, van Dongen JJ, Staal FJ . New insights and unresolved issues regarding insertional mutagenesis in X-linked SCID gene therapy. Mol Ther 2007; 15: 1910–1916.

4. Vetrie D, Vorechovsky I, Sideras P, Holland J, Davies A, Flinter F et al. The gene involved in X-linked agammaglobulinaemia is a member of the src family of protein-tyrosine kinases. Nature 1993; 361: 226–233.

5. Tsukada S, Saffran DC, Rawlings DJ, Parolini O, Allen RC, Klisak I et al. Deficient expression of a B cell cytoplasmic tyrosine kinase in human X-linked agammaglobulinemia. Cell 1993; 72: 279–290.

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