Leukemic fusion genes MLL/AF4 and AML1/MTG8 support leukemic self-renewal by controlling expression of the telomerase subunit TERT-Reference-Cited by-同舟云学术

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Leukemic fusion genes MLL/AF4 and AML1/MTG8 support leukemic self-renewal by controlling expression of the telomerase subunit TERT

Published:2010-08-05 Issue:10 Volume:24 Page:1751-1759
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Gessner A,Thomas M,Garrido Castro P,Büchler L,Scholz A,Brümmendorf T H,Martinez Soria N,Vormoor J,Greil J,Heidenreich O

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/leu2010155.pdf

Reference59 articles.

1. Dick JE . Stem cell concepts renew cancer research. Blood 2008; 112: 4793–4807.

2. Misaghian N, Ligresti G, Steelman LS, Bertrand FE, Basecke J, Libra M et al. Targeting the leukemic stem cell: the Holy Grail of leukemia therapy. Leukemia 2009; 23: 25–42.

3. Deng Y, Chan SS, Chang S . Telomere dysfunction and tumour suppression: the senescence connection. Nat Rev 2008; 8: 450–458.

4. Nakamura TM, Morin GB, Chapman KB, Weinrich SL, Andrews WH, Lingner J et al. Telomerase catalytic subunit homologs from fission yeast and human. Science 1997; 277: 955–959.

5. Lin SY, Elledge SJ . Multiple tumor suppressor pathways negatively regulate telomerase. Cell 2003; 113: 881–889.

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