Cellular prion protein mediates impairment of synaptic plasticity by amyloid-β oligomers
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/nature07761.pdf
Reference37 articles.
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3. Cleary, J. P. et al. Natural oligomers of the amyloid-β protein specifically disrupt cognitive function. Nature Neurosci. 8, 79–84 (2005)
4. Chromy, B. A. et al. Self-assembly of Aβ1–42 into globular neurotoxins. Biochemistry 42, 12749–12760 (2003)
5. Lacor, P. N. et al. Abeta oligomer-induced aberrations in synapse composition, shape, and density provide a molecular basis for loss of connectivity in Alzheimer’s disease. J. Neurosci. 27, 796–807 (2007)
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