Frequent inactivation of A20 in B-cell lymphomas-Reference-Cited by-同舟云学术

Frequent inactivation of A20 in B-cell lymphomas

Published:2009-05-03 Issue:7247 Volume:459 Page:712-716
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Kato Motohiro,Sanada Masashi,Kato Itaru,Sato Yasuharu,Takita Junko,Takeuchi Kengo,Niwa Akira,Chen Yuyan,Nakazaki Kumi,Nomoto Junko,Asakura Yoshitaka,Muto Satsuki,Tamura Azusa,Iio Mitsuru,Akatsuka Yoshiki,Hayashi Yasuhide,Mori Hiraku,Igarashi Takashi,Kurokawa Mineo,Chiba Shigeru,Mori Shigeo,Ishikawa Yuichi,Okamoto Koji,Tobinai Kensei,Nakagama Hitoshi,Nakahata Tatsutoshi,Yoshino Tadashi,Kobayashi Yukio,Ogawa Seishi

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature07969.pdf

Reference29 articles.

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2. Song, H. Y., Rothe, M. & Goeddel, D. V. The tumor necrosis factor-inducible zinc finger protein A20 interacts with TRAF1/TRAF2 and inhibits NF-κB activation. Proc. Natl Acad. Sci. USA 93, 6721–6725 (1996)

3. Lee, E. G. et al. Failure to regulate TNF-induced NF-κB and cell death responses in A20-deficient mice. Science 289, 2350–2354 (2000)

4. Boone, D. L. et al. The ubiquitin-modifying enzyme A20 is required for termination of Toll-like receptor responses. Nature Immunol. 5, 1052–1060 (2004)

5. Wang, Y. Y., Li, L., Han, K. J., Zhai, Z. & Shu, H. B. A20 is a potent inhibitor of TLR3- and Sendai virus-induced activation of NF-κB and ISRE and IFN-β promoter. FEBS Lett. 576, 86–90 (2004)

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