Myelin failure in multiple sclerosis: Breaking the spell of Notch
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
http://www.nature.com/articles/nm1002-1075.pdf
Reference13 articles.
1. Lassmann, H., Brück, W. & Lucchinetti, C. Heterogeneity of multiple sclerosis pathogenesis: Implications for diagnosis and therapy. Trends Mol. Med. 7, 115–121 (2001)
2. John, R. et al. Multiple sclerosis: Re-expression of a developmental pathway that restricts oligodendrocyte maturation. Nature Med. 8, 1115–1121 (2002).
3. Wolswijk, G. Oligodendrocyte precursor cells in the demyelinated multiple sclerosis spinal cord. Brain 125, 338–349 (2002).
4. Franklin, R.J.M. Why does remyelination fail in multiple sclerosis? Nature Rev. Neurosci. 3, 705–714 (2002).
5. Chang, A., Tourtellotte, W.W., Rudick, R. & Trapp, B.D. Premyelinating oligodendrocytes in chronic lesions of multiple sclerosis. N. Engl. J. Med. 346, 165–173 (2002).
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1. An enhanced triple fluorescence flow-cytometry-based assay shows differential activation of the Notch signaling pathway by human papillomavirus E6 proteins;Scientific Reports;2022-02-22
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3. Low levels of cobalamin, epidermal growth factor, and normal prions in multiple sclerosis spinal cord;Neuroscience;2015-07
4. An Overview of Notch Signaling in Adult Tissue Renewal and Maintenance;Current Alzheimer Research;2012-02-01
5. Indirubin-3′-monoxime, a derivative of a Chinese anti-leukemia medicine, inhibits Notch1 signaling;Cancer Letters;2008-07
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