Alternatively spliced, truncated GCSF receptor promotes leukemogenic properties and sensitivity to JAK inhibition-Reference-Cited by-同舟云学术

Alternatively spliced, truncated GCSF receptor promotes leukemogenic properties and sensitivity to JAK inhibition

Published:2013-10-30 Issue:5 Volume:28 Page:1041-1051
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Mehta H M,Futami M,Glaubach T,Lee D W,Andolina J R,Yang Q,Whichard Z,Quinn M,Lu H F,Kao W M,Przychodzen B,Sarkar C A,Minella A^ORCID,Maciejewski J P,Corey S J

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/leu2013321.pdf

Reference45 articles.

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2. Liu F, Poursine-Laurent J, Link DC . The granulocyte colony-stimulating factor receptor is required for the mobilization of murine hematopoietic progenitors into peripheral blood by cyclophosphamide or interleukin-8 but not flt-3 ligand. Blood 1997; 90: 2522–2528.

3. Dong F, Hoefsloot LH, Schelen AM, Broeders CA, Meijer Y, Veerman AJ et al. Identification of a nonsense mutation in the granulocyte-colony-stimulating factor receptor in severe congenital neutropenia. Proc Natl Acad Sci USA 1994; 91: 4480–4484.

4. Dong F, Brynes RK, Tidow N, Welte K, Lowenberg B, Touw IP . Mutations in the gene for the granulocyte colony-stimulating-factor receptor in patients with acute myeloid leukemia preceded by severe congenital neutropenia. N Engl J Med 1995; 333: 487–493.

5. Germeshausen M, Ballmaier M, Welte K . Incidence of CSF3R mutations in severe congenital neutropenia and relevance for leukemogenesis: Results of a long-term survey. Blood 2007; 109: 93–99.

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