STIM1 signalling controls store-operated calcium entry required for development and contractile function in skeletal muscle-Reference-Cited by-同舟云学术

STIM1 signalling controls store-operated calcium entry required for development and contractile function in skeletal muscle

Published:2008-05-18 Issue:6 Volume:10 Page:688-697
ISSN:1465-7392
Container-title:Nature Cell Biology
language:en
Short-container-title:Nat Cell Biol

Author:

Stiber Jonathan,Hawkins April,Zhang Zhu-Shan,Wang Sunny,Burch Jarrett,Graham Victoria,Ward Cary C.,Seth Malini,Finch Elizabeth,Malouf Nadia,Williams R. Sanders,Eu Jerry P.,Rosenberg Paul

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology

Link

http://www.nature.com/articles/ncb1731.pdf

Reference50 articles.

1. Liou, J. et al. STIM is a Ca2+ sensor essential for Ca2+-store-depletion-triggered Ca2+ influx. Curr. Biol. 15, 1235–1241 (2005).

2. Roos, J. et al. STIM1, an essential and conserved component of store-operated Ca2+ channel function. J. Cell Biol. 169, 435–445 (2005).

3. Zhang, S. L. et al. STIM1 is a Ca2+ sensor that activates CRAC channels and migrates from the Ca2+ store to the plasma membrane. Nature 437, 902–905 (2005).

4. Luik, R. M., Wu, M. M., Buchanan, J. & Lewis, R. S. The elementary unit of store-operated Ca2+ entry: local activation of CRAC channels by STIM1 at ER-plasma membrane junctions. J. Cell Biol. 174, 815–825 (2006).

5. Wu, M. M., Buchanan, J., Luik, R. M. & Lewis, R. S. Ca2+ store depletion causes STIM1 to accumulate in ER regions closely associated with the plasma membrane. J. Cell Biol. 174, 803–813 (2006).

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