Specialized interfaces of Smc5/6 control hinge stability and DNA association-Reference-Cited by-同舟云学术

Specialized interfaces of Smc5/6 control hinge stability and DNA association

Published:2017-01-30 Issue:1 Volume:8 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Alt Aaron,Dang Hung Q.,Wells Owen S.,Polo Luis M.,Smith Matt A.,McGregor Grant A.,Welte Thomas,Lehmann Alan R.,Pearl Laurence H.^ORCID,Murray Johanne M.,Oliver Antony W.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms14011.pdf

Reference67 articles.

1. Lehmann, A. R. et al. The rad18 gene of Schizosaccharomyces pombe defines a new subgroup of the SMC superfamily involved in DNA repair. Mol. Cell Biol. 15, 7067–7080 (1995).

2. Ju, L. et al. SMC6 is an essential gene in mice, but a hypomorphic mutant in the ATPase domain has a mild phenotype with a range of subtle abnormalities. DNA Repair (Amst) 12, 356–366 (2013).

3. Ampatzidou, E., Irmisch, A., O'Connell, M. J. & Murray, J. M. Smc5/6 is required for repair at collapsed replication forks. Mol. Cell Biol. 26, 9387–9401 (2006).

4. Branzei, D. et al. Ubc9- and mms21-mediated sumoylation counteracts recombinogenic events at damaged replication forks. Cell 127, 509–522 (2006).

5. De Piccoli, G. et al. Smc5-Smc6 mediate DNA double-strand-break repair by promoting sister-chromatid recombination. Nat. Cell Biol. 8, 1032–1034 (2006).

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