Mechanisms of RIPK3‐induced inflammation
Author:
Affiliation:
1. Department of Clinical Microbiology and Immunology, Sackler Faculty of Medicine, Tel Aviv University Tel Aviv Israel
Funder
Israel Science Foundation
Recanati Foundation
Varda and Boaz Dotan Research Center
Publisher
Wiley
Subject
Cell Biology,Immunology,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1038/icb.2016.124
Reference82 articles.
1. RIP3, a Novel Apoptosis-inducing Kinase
2. Identification of RIP3, a RIP-like kinase that activates apoptosis and NFκB
3. Mouse Receptor Interacting Protein 3 Does Not Contain a Caspase-Recruiting or a Death Domain but Induces Apoptosis and Activates NF-κB
4. RIP3, an Energy Metabolism Regulator That Switches TNF-Induced Cell Death from Apoptosis to Necrosis
5. Mixed Lineage Kinase Domain-like Protein Mediates Necrosis Signaling Downstream of RIP3 Kinase
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