Dual PI3K/mTOR inhibitor BEZ235 as a promising therapeutic strategy against paclitaxel-resistant gastric cancer via targeting PI3K/Akt/mTOR pathway-Reference-Cited by-同舟云学术

Dual PI3K/mTOR inhibitor BEZ235 as a promising therapeutic strategy against paclitaxel-resistant gastric cancer via targeting PI3K/Akt/mTOR pathway

Published:2018-01-26 Issue:2 Volume:9 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Chen Dongshao,Lin Xiaoting,Zhang Cheng,Liu Zhentao,Chen Zuhua,Li Zhongwu,Wang Jingyuan,Li Beifang,Hu Yanting,Dong Bin,Shen Lin,Ji Jiafu,Gao Jing,Zhang Xiaotian

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-017-0132-2.pdf

Reference45 articles.

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3. Gong, J. et al. The multicenter, phase II prospective study of paclitaxel plus capecitabine as first-line chemotherapy in advanced gastric carcinoma. Oncologist 19, 173–174 (2014).

4. Lu, M., Gao, J., Wang, X. C. & Shen, L. Expressions of thymidylate synthase, thymidine phosphorylase, class III beta-tubulin, and excision repair cross-complementing group 1 predict response in advanced gastric cancer patients receiving capecitabine plus paclitaxel or cisplatin. Chin. J. Cancer Res 23, 288–294 (2011).

5. Gao, J., Lu, M., Yu, J. W., Li, Y. Y. & Shen, L. Thymidine phosphorylase/beta-tubulin III expressions predict the response in Chinese advanced gastric cancer patients receiving first-line capecitabine plus paclitaxel. BMC Cancer 11, 177 (2011).

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