Arsenic circumvents the gefitinib resistance by binding to P62 and mediating autophagic degradation of EGFR in non-small cell lung cancer-Reference-Cited by-同舟云学术

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Arsenic circumvents the gefitinib resistance by binding to P62 and mediating autophagic degradation of EGFR in non-small cell lung cancer

Published:2018-09-20 Issue:10 Volume:9 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Mao Jianhua,Ma Lie,Shen Yan,Zhu Kongkai,Zhang Ru,Xi Wenda,Ruan Zheng,Luo Cheng,Chen Zhu,Xi Xiaodong,Chen Saijuan

Funder

National Natural Science Foundation of China

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-018-0998-7.pdf

Reference43 articles.

1. Siegel, R. L., Miller, K. D. & Jemal, A. Cancer statistics. CA Cancer J. Clin. 66, 7–30 (2016).

2. Herbst, R. S., Heymach, J. V. & Lippman, S. M. Lung cancer. N. Engl. J. Med. 359, 1367–1380 (2008).

3. Ramalingam, S. S., Owonikoko, T. K. & Khuri, F. R. Lung cancer: new biological insights and recent therapeutic advances. CA Cancer J. Clin. 61, 91–112 (2011).

4. Sharma, S. V., Bell, D. W., Settleman, J. & Haber, D. A. Epidermal growth factor receptor mutations in lung cancer. Nat. Rev. Cancer 7, 169–181 (2007).

5. Mok, T. S. et al. Gefitinib or carboplatin-paclitaxel in pulmonary adenocarcinoma. N. Engl. J. Med. 361, 947–957 (2009).

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