TLR9 is essential for HMGB1-mediated post-myocardial infarction tissue repair through affecting apoptosis, cardiac healing, and angiogenesis-Reference-Cited by-同舟云学术

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TLR9 is essential for HMGB1-mediated post-myocardial infarction tissue repair through affecting apoptosis, cardiac healing, and angiogenesis

Published:2019-06-17 Issue:7 Volume:10 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Liu Fang-Yuan,Fan Di,Yang Zheng,Tang Nan,Guo Zhen,Ma Shu-Qing,Ma Zhen-Guo,Wu Hai-Ming,Deng Wei,Tang Qi-Zhu

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-019-1718-7.pdf

Reference45 articles.

1. Teringova, E. & Tousek, P. Apoptosis in ischemic heart disease. J. Transl. Med. 15, 87 (2017).

2. Moe, G. W. & Mar I N-Garc I A, J. E. Role of cell death in the progression of heart failure. Heart Fail. Rev. 21, 157–167 (2016).

3. Sousa, F. M., Abd, J. A., Stannard, G. A. & Heather, L. C. Hypoxia-inducible factor 1 signalling, metabolism and its therapeutic potential in cardiovascular disease. Biochim. Biophys. Acta Mol. Basis Dis. 1865, 831–843 (2019).

4. Ohto, U. et al. Toll-like receptor 9 contains two DNA binding sites that function cooperatively to promote receptor dimerization and activation. Immunity 48, 649–658.e4 (2018).

5. Yang, H., Wang, H., Chavan, S. S. & Andersson, U. High mobility group box protein 1 (HMGB1): the prototypical endogenous danger molecule. Mol. Med. 21(Suppl. 1), S6–S12 (2015).

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