Loss of Drosha underlies dopaminergic neuron toxicity in models of Parkinson’s disease-Reference-Cited by-同舟云学术

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Loss of Drosha underlies dopaminergic neuron toxicity in models of Parkinson’s disease

Published:2018-06 Issue:6 Volume:9 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Wang Ronglin,Lu Fangfang,Zhu Gang,Feng Dayun,Nie Tiejian,Tao Kai,Yang Shaosong,Lei Jie,Huang Lu,Mao Zixu,Yang Qian

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-018-0716-5.pdf

Reference55 articles.

1. Abeliovich, A. & Gitler, A. D. Defects in trafficking bridge Parkinson’s disease pathology and genetics. Nature 539, 207–216 (2016).

2. Yang, Q. et al. Regulation of neuronal survival factor MEF2D by chaperone-mediated autophagy. Science 323, 124–127 (2009).

3. Yu, C., Kim, B. S. & Kim, E. FAF1 mediates regulated necrosis through PARP1 activation upon oxidative stress leading to dopaminergic neurodegeneration. Cell Death Differ. 23, 1873–1885 (2016).

4. Wu, R. et al. c-Abl-p38alpha signaling plays an important role in MPTP-induced neuronal death. Cell Death Differ. 23, 542–552 (2016).

5. Gao, L. et al. Oxidation of survival factor MEF2D in neuronal death and Parkinson’s disease. Antioxid. Redox Signal. 20, 2936–2948 (2014).

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