Preconditioning, induced by sub-toxic dose of the neurotoxin L-BMAA, delays ALS progression in mice and prevents Na+/Ca2+ exchanger 3 downregulation-Reference-Cited by-同舟云学术

Preconditioning, induced by sub-toxic dose of the neurotoxin L-BMAA, delays ALS progression in mice and prevents Na+/Ca2+ exchanger 3 downregulation

Published:2018-02 Issue:2 Volume:9 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Anzilotti Serenella,Brancaccio Paola,Simeone Giuseppe,Valsecchi Valeria,Vinciguerra Antonio,Secondo Agnese^ORCID,Petrozziello Tiziana,Guida Natascia,Sirabella Rossana,Cuomo Ornella,Cepparulo Pasquale,Herchuelz Andrè,Amoroso Salvatore,Di Renzo Gianfranco,Annunziato Lucio,Pignataro Giuseppe^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-017-0227-9.pdf

Reference39 articles.

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2. Stenzel-Poore, M. P. et al. Effect of ischaemic preconditioning on genomic response to cerebral ischaemia: similarity to neuroprotective strategies in hibernation and hypoxia-tolerant states. Lancet 362, 1028–1037 (2003).

3. Karamyan, V. T. & Speth, R. C. Animal models of BMAA neurotoxicity: a critical review. Life Sci. 82, 233–246 (2008).

4. Delzor, A. et al. Searching for a link between the l-BMAA neurotoxin and amyotrophic lateral sclerosis: a study protocol of the French BMAALS programme. BMJ Open 4, e005528 (2014).

5. Delzor, A., Marin, B., Boumediene, F., Preux, P. M. & Couratier, P. BMAALS: a French national project searching for a link between amyotrophic lateral sclerosis and the neurotoxic amino acid l-BMAA. Amyotroph. Lateral Scler. Frontotemporal Degener. 15, 155–156 (2014).

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