Podocyte-specific Rac1 deficiency ameliorates podocyte damage and proteinuria in STZ-induced diabetic nephropathy in mice-Reference-Cited by-同舟云学术

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Podocyte-specific Rac1 deficiency ameliorates podocyte damage and proteinuria in STZ-induced diabetic nephropathy in mice

Published:2018-03 Issue:3 Volume:9 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Lv Zhimei,Hu Mengsi,Fan Minghua,Li Xiaobing,Lin Jiangong,Zhen Junhui,Wang Ziyang,Jin Haijun,Wang Rong

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-018-0353-z.pdf

Reference46 articles.

1. Kanwar, Y. S., Sun, L., Xie, P., Liu, F. Y. & Chen, S. A glimpse of various pathogenetic mechanisms of diabetic nephropathy. Annu. Rev. Pathol. 6, 395–423 (2011).

2. Reiser, J. & Sever, S. Podocyte biology and pathogenesis of kidney disease. Annu. Rev. Med 64, 357–366 (2013).

3. Brinkkoetter, P. T., Ising, C. & Benzing, T. The role of the podocyte in albumin filtration. Nat. Rev. Nephrol. 9, 328–336 (2013).

4. Reidy, K. & Suszta, K. Epithelial-mesenchymal transition and podocyte loss in diabetic kidney disease. Am. J. Kidney Dis. 54, 590–593 (2009).

5. Bid, H. K., Roberts, R. D., Manchanda, P. K. & Houghton, P. J. RAC1: an emerging therapeutic option for targeting cancer angiogenesis and metastasis. Mol. Cancer Ther. 12, 1925–1934 (2013).

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