Dysfunctional autophagy following exposure to pro-inflammatory cytokines contributes to pancreatic β-cell apoptosis-Reference-Cited by-同舟云学术

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Dysfunctional autophagy following exposure to pro-inflammatory cytokines contributes to pancreatic β-cell apoptosis

Published:2018-01-24 Issue:2 Volume:9 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Lambelet Martine,Terra Leticia F.,Fukaya Makiko,Meyerovich Kira,Labriola Leticia,Cardozo Alessandra K.,Allagnat Florent

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-017-0121-5.pdf

Reference54 articles.

1. Patterson, C. C., Dahlquist, G. G., Gyurus, E., Green, A. & Soltesz, G. Incidence trends for childhood type 1 diabetes in Europe during 1989-2003 and predicted new cases 2005-20: a multicentre prospective registration study. Lancet 373, 2027–2033 (2009).

2. Meyerovich, K., Ortis, F., Allagnat, F. & Cardozo, A. K. Endoplasmic reticulum stress and the unfolded protein response in pancreatic islet inflammation. J. Mol. Endocrinol. 57, R1–R17 (2016).

3. Dolman, N. J., Chambers, K. M., Mandavilli, B., Batchelor, R. H. & Janes, M. S. Tools and techniques to measure mitophagy using fluorescence microscopy. Autophagy 9, 1653–1662 (2013).

4. Cebollero, E., Reggiori, F. & Kraft, C. Reticulophagy and ribophagy: regulated degradation of protein production factories. Int. J. Cell Biol. 2012, 182834 (2012).

5. Lee, M. S. Role of islet beta cell autophagy in the pathogenesis of diabetes. Trends Endocrinol. Metab. 25, 620–627 (2014).

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