CCR4-dependent reduction in the number and suppressor function of CD4+Foxp3+ cells augments IFN-γ-mediated pulmonary inflammation and aggravates tuberculosis pathogenesis-Reference-Cited by-同舟云学术

CCR4-dependent reduction in the number and suppressor function of CD4+Foxp3+ cells augments IFN-γ-mediated pulmonary inflammation and aggravates tuberculosis pathogenesis

Published:2018-12-21 Issue:1 Volume:10 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Bertolini Thais B.,Piñeros Annie R.,Prado Rafael Q.,Gembre Ana Flávia,Ramalho Leandra N. Z.,Alves-Filho José Carlos^ORCID,Bonato Vânia L. D.

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-018-1240-3.pdf

Reference46 articles.

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2. Tornheim, J. A. & Dooley, K. E. Tuberculosis associated with HIV infection. Microbiol. Spectr. 5, https://doi.org/10.1128/microbiolspec.TNMI7-0028-2016 (2017).

3. WHO. (2017).

4. Flynn, J. L., Chan, J. & Lin, P. L. Macrophages and control of granulomatous inflammation in tuberculosis. Mucosal Immunol. 4, 271–278 (2011).

5. O’Garra, A. et al. The immune response in tuberculosis. Annu Rev. Immunol. 31, 475–527 (2013).

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