Soluble TNF-like weak inducer of apoptosis (TWEAK) enhances poly(I:C)-induced RIPK1-mediated necroptosis-Reference-Cited by-同舟云学术

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Soluble TNF-like weak inducer of apoptosis (TWEAK) enhances poly(I:C)-induced RIPK1-mediated necroptosis

Published:2018-10-22 Issue:11 Volume:9 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Anany Mohamed A.,Kreckel Jennifer,Füllsack Simone,Rosenthal Alevtina,Otto Christoph,Siegmund Daniela,Wajant Harald^ORCID

Funder

Deutsche Forschungsgemeinschaft

Deutsche Krebshilfe

M. A. is a German Egyptian Research Long Term Scholarship (GERLS) holder funded by DAAD.

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-018-1137-1.pdf

Reference52 articles.

1. Wajant, H. The TWEAK-Fn14 system as a potential drug target. Br. J. Pharmacol. 170, 748–764 (2013).

2. Wu., F. et al. TNF-like weak inducer of apoptosis (TWEAK) promotes beta cell neogenesis from pancreatic ductal epithelium in adult mice. PLoS ONE 8, e72132 (2013).

3. Karaca, G. et al. TWEAK/Fn14 signaling is required for liver regeneration after partial hepatectomy in mice. PLoS ONE 9, e83987 (2014).

4. Girgenrath, M. et al. TWEAK, via its receptor Fn14, is a novel regulator of mesenchymal progenitor cells and skeletal muscle regeneration. EMBO J. 25, 5826–5839 (2006).

5. Chicheportiche, Y. et al. TWEAK, a new secreted ligand in the tumor necrosis factor family that weakly induces apoptosis. J. Biol. Chem. 272, 32401–32410 (1997).

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